Publications of Marina Gumenscheimer
All genres
Journal Article (10)
2012
Journal Article
7, p. e39155 - e39155 (2012)
TLR9-Dependent and Independent Pathways Drive Activation of the Immune System by Propionibacterium Acnes. PLoS ONE 2007
Journal Article
196, pp. 79 - 88 (2007)
Stage of primary infection with lymphocytic choriomeningitis virus determines predisposition or resistance of mice to secondary bacterial infections. Medical Microbiology and Immunology 2005
Journal Article
174, pp. 4295 - 4300 (2005)
Requirement for TLR9 in the Immunomodulatory Activity of Propionibacterium acnes1. The Journal of Immunology
Journal Article
11, pp. 3398 - 3404 (2005)
Lethal effect and apoptotic DNA fragmentation in response of D-GalN-treated mice to bacterial LPS can be suppressed by pre-exposure to minute amount of bacterial LPS: Dual role of TNF receptor 1. World Journal of Gastroenterology 2003
Journal Article
71 (8), pp. 4456 - 4462 (2003)
Toll-Like Receptor 2- and 6-Mediated Stimulation by Macrophage-Activating Lipopeptide 2 Induces Lipopolysaccharide (LPS) Cross Tolerance in Mice, Which Results in Protection from Tumor Necrosis Factor Alpha but in Only Partial Protection from Lethal LPS Doses. Infection and Immunity
Journal Article
9 (5), pp. 308 - 312 (2003)
Role of interferons in LPS hypersensitivity. Journal of Endotoxin Research
Journal Article
24 (3), pp. 193 - 198 (2003)
A striking correlation between lethal activity and apoptotic DNA fragmentation of liver in response of D-galactosamine- sensitized mice to a non-lethal amount of lipopolysaccharide. Acta Pharmacologica Sinica 2002
Journal Article
70 (10), pp. 5596 - 5603 (2002)
Beneficial or Deleterious Effects of a Preexisting Hypersensitivity to Bacterial Components on the Course and Outcome of Infection. Infection and Immunity
Journal Article
8 (5), pp. 319 - 327 (2002)
The role of the liver in the response to LPS: experimental and clinical findings. Journal of Endotoxin Research 2001
Journal Article
3 (14-15), pp. 1213 - 1222 (2001)
Role of lipopolysaccharide susceptibility in the innate immune response to Salmonella typhimurium infection: LPS, a primary target for recognition of Gram-negative bacteria. Microbes and Infection Thesis - PhD (1)
2003
Thesis - PhD
Bacteria- or virus-induced hypersensitivity to LPS and other bacterial components. Dissertation, Universita'Degli Studi di Bari, Bari, Italy (2003)